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When the body is under excessive stress, these proteins exert a protective role by lessening oxidative action of the reactive oxygen species (ROS) and a wide range of metabolic stress, including structural and functional myodamage [87, 88]

Further, by examining hippocampal synaptosomes containing cytosolic and mitochondrial machinery during epileptogenesis, it has been observed that there is reduced ATP-linked baseline respiration of affected neurons during the acute and chronic phases of epileptogenesis

These numbers were unprecedented when first published and set the benchmark that all subsequent obesity treatments have been measured against