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Nakamura, M

Additional contributing factors include increased expression of insulin-like growth factor 1 (IGF1) receptors, IGF1-binding protein (which protects IGF1 from proteolysis) in Purkinje cells [8], as well as upregulated mRNA expression of GLUT1 and GLUT3 [80]

Since PPAR- agonistic drugs such as IRB are responsible for strong organ-protective effects, including anti-fibrotic, anti-oxidant, and anti-inflammatory effects, IRB was proven to be effective in protection from CP-induced ovarian injury (Wang et al

Regulation of cell proliferation and apoptosis: Glutathione depletion is a signal for apoptosis (programmed cell death), while adequate levels support normal cell proliferation [2][5]