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In part, this can be explained by lower phagocytic uptake of mutants, which can impede traversal of the blood-brain barrier via a Trojan horse mechanism a tactic by which C

Glutathione S transferase and catalase gene polymorphisms did not tend to influence the severity of hemoglobin E/-thalassemia Main Article Content Abstract Background Thalassemia, a monogenic genetic disease of red blood cells, is spread widely throughout the world

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