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Collectively, ACSL4-dependent ferroptosis in alveolar epithelial type II cells facilitates Bet v 1-induced T H 2/T H 17-related inflammatory and epithelial damage, orchestrating allergic disease and being capable of acting as an intervention target for allergy therapy

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Statistics and reproducibility (De-)acetylation assays as well as SUMO2/ubiquitin cleavage assays were performed in at least two independent experiments as indicated with similar results obtained

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