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Glabridin exhibited therapeutic potential in the treatment of DN through improving renal function by repressing not only ferroptosis but also VEGF/p-Akt/p-ERK1/2 axis activation [247]

Additional studies are needed in order to evaluate potential compensatory activation of PPAR upon inhibition of PGC1 as well as to elucidate whether long-term tesaglitazar treatment causes irreversible cardiac dysfunction and remodeling, accompanied by increased expression of heart failure markers

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These injectable peptides are not approved by the United States Food and Drug Administration (FDA), and there is no FDA oversight of their manufacturing for consumer use