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In addition, recent studies suggest that under certain conditions, such as hyperreduced CoQ pools and elevated mitochondrial membrane potential, reverse electron transport may occurwhereby electrons flow from ubiquinol (CoQ 10 H 2 ) back to complex I, generating NADH 110,111

These clinical symptoms affect patients quality of life to varying degrees

Further specificity may be the noted special interaction with NO-system and NO-agents in various models and species [7,8,14] since BPC 157 induced the NO-release of its own [78,79]