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In the presence of inflammation, alterations in intestinal barrier function, along with increased intestinal permeability and bacterial translocation, can trigger IBD and autoimmunity in genetically predisposed individuals [68, 69]

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Synaptotagmin-binding cytoplasmic RNA-interacting protein (SYNCRIP), also known as heterogeneous nuclear ribonucleoprotein Q (hnRNP Q), is an RBP implicated in diverse cellular processes [13]

Nevertheless, redox cycling remains an understated mechanism of toxicity due to the fleeting existence of free radical intermediates which hinders their quantification in real time, both in vitro and in vivo 6