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Research indicates that NAC can regualate autophagy and promote the clearance of -amyloid proteins [258], an effect that may relate to its capacity to scavenge one-electron oxidants [259], thereby positioning NAC as a potential therapeutic strategy for Alzheimers disease

These results provide biochemical evidence that GSK3-mediated phosphorylation is required for the direct physical interaction and subsequent ubiquitination of ETV6 by the SCF FBXW7 complex

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Endothelial dysfunction, characterized by damage to the cells lining the micro vessels, reduces their ability to regulate blood flow and permeability (Sukriti et al., 2014)