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Conversely, excessive NADPH accumulation can induce reductive stress, leading to the upregulation of NADPH oxidase 4 through activation of the PI3K/Akt signaling pathway, thereby promoting ROS generation, as observed in osteoarthritis (96)

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This growing body of evidence underscores the therapeutic potential of targeting the gut microbiota and immune pathways to develop novel interventions for neurological diseases

Glutathione may interact with certain medications metabolized via glutathione conjugation, and excessive antioxidant activity can theoretically blunt beneficial inflammatory signaling during metabolic adaptation