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Description

Barrier disruptions increase gut leakiness and attract white blood cells to perpetuate local inflammation that damages the gut mucosal barrier and underlying tissues

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In the structure of Ontak, IL-2 and diphtheria toxin are responsible for targeting and toxic activity, respectively (158)

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Clonal evolution and treatment outcomes in hematopoietic neoplasms arising in patients with germline RUNX1 mutations

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