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L.IameleL.GoughJ.et al (2003)

Recent investigations further indicated that UA may operate as a protective factor mediated through astroglia for dopaminergic neurons from glutamate toxicity ( + -induced depolarization amplifies neuroprotection provided by UA through a mechanism involving Ca 2 elevation and extracellular signal-regulated kinases (ERK 1/2 ) activation ( Figure 3 )

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(2008) demonstrated that an alternative pathway of glutathione degradation by means of gamma-glutamyl cyclotransferase (GGCT) and 5-oxo-prolinase (5Opase) dominates over GGT degradation in plant tissues