Further, although several previous studies have evaluated GSH levels in various extracellular compartments, including bronchoalveolar lavage (BAL) fluid, these studies generally focused on alterations in GSH:GSSG balance in association with disease, or the consumption of GSH during prostaglandin or leukotriene synthesis, with more modest phenotypic, pathophysiologic, or mechanistic information (4, 5)
Therefore, noting the lack of spontaneous oxidation of free GSH between the two EDTA-treated samples stored at 80 o C, this result also suggests that the differences in GSH between the heparinized plasma (Preparation Condition 1) and deproteinized EDTA-treated plasma (Preparation Condition 2) are due to spontaneous oxidation of free GSH occurring in the heparinized plasma prior to the deproteinization process
Its been smooth sailing and Im sure it will always be
doi: 10.1111/jgs.15767 541 National Institute for Health and Care Excellence - NICE, Chronic fatigue syndrome/myalgic encephalomyelitis (or encephalopathy): diagnosis and management (2007)
