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(2011), however, suggest that the constitutive activity of GHSR1a is not the result of dimerization

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In a rat model simulating SAM conditions, ~ 24% of hepatic genes were differentially expressed with upregulation of TRIB3 and FGF21 (markers of disrupted hepatic signaling) and long-lasting steatosis was attributed to peroxisome loss via transcription-mediated pexophagy [137]