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Because free-radical damage is in itself a major contributor the aging process, this age-related decrease in glutathione production can lead to a self-perpetuating cycle of mental and physical decline

[Neurochemical perspectives of the function of monoamine oxidases]

[8] The age-related decline and its implications The decline of circulating GHK from 200 ng/mL at age 20 to 80 ng/mL by age 60 correlates with reduced wound healing capacity, thinner skin, decreased collagen density, and slower tissue repair

Studies in animal models have highlighted the therapeutic potential of neurotrophic mimetics to enhance adult neurogenesis as per their bioavailability, specificity, and minimal side effects