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For example, recent studies have shown that ER stress within beta cells leads to translational errors in the reading frame of human insulin mRNA, resulting in the generation of defective polypeptides and the formation of neo-epitopes, capable of recognition by circulating T cell populations which have escaped prior thymic deletion (Kracht et al
1B), and it may be that declining substrate availability is rate-limiting to NAD(P) + flux, providing a mechanism for the impact of NMN treatment on ovarian function and fertility
monocytogenes is able to synthesize GSH and to import host-derived GSH as adaptation strategy under infection conditions (Reniere et al., 2015)
