glutathione structure molecule and mechanism to form Chemical of (GSH), a tripeptide made up of Glutathione

Organoids derived from ectopic endometriosis lesions have shown promising utility in modeling disease-specific characteristics, including the altered expression of ECM-receptor interaction genes ( COL3A1 , FN1 , ITGA11 , LAMA2 ), WNT-pathway genes ( CTNNA2 , LEF1 , WNT11) and hormone-responsive genes ( HSD11B1 , LIFR , PGR , PRLR ) [40, 55, 64]

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Guo, Q., Goldenberg, J

Platforms obtained in this manner retain the specificity of TME-oriented design yet remain relatively compatible with existing pathways for manufacturing, quality control and safety assessment in drug development [61,62,63]