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In this paper, we will review recent studies on the roles of Rho proteins in the regulation of hippocampal long-term potentiation (LTP) and long-term depression (LTD), the most extensively studied forms of synaptic plasticity widely regarded as cellular mechanisms for learning and memory

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GHK-Cu modulates the expression of over 4,000 human genes (31.2% of the genome), activating 1,942 genes (including 47 DNA repair genes) and suppressing 762 genes

6,7,8,9 Canonical enzymatic L-lactylation uses lactyl-coenzyme A (lactyl-CoA) as a precursor of the L-lactyl moiety, followed by covalent attachment of the lactyl group to lysine residues via lactyltransferases