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Blocking the cytokine-mediated inflammatory cell death signaling pathway may benefit patients with COVID-19 or other infectious diseases by limiting tissue damage ( Figure 1 Implications on Antibacterial and Antiviral Immunity According to in vitro studies, TNF- facilitates the SARS-CoV interaction with angiotensin-converting enzyme 2 (ACE2) (60)

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These results suggest that GpoA is critical not only for resistance to oxidative stress but also for effective invasion and systemic dissemination during infection