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Alternatively, alterations in non-mitochondrial genes not uncommonly associated with ASD such as PTEN [20] and mTOR [21, 22] can alter mitochondrial function, potentially contributing to mitochondrial dysfunction and predisposing the mitochondrial to be sensitive to environmental agents

The goal is to find your personal optimal dose, the highest amount you can tolerate without significant issues while achieving your desired results

Mitochondrial dysfunction may explain symptom variation in Phelan-McDermid Syndrome

Anti-epileptic activity of daidzin in PTZ-induced mice model by targeting oxidative stress and BDNF/VEGF signaling