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The compound appears to work by increasing intracellular NAD+ levels (through preventing nicotinamide consumption by NNMT), which activates sirtuins and enhances mitochondrial function and fatty acid oxidation, while also promoting a phenomenon called "browning" of white adipose tissue - the transformation of energy-storing white fat cells into metabolically active beige/brown adipocytes that burn fatty acids for thermogenesis rather than storing them
[1] The Human Evidence Gap and Where Research Is Heading The most intellectually honest statement about BPC-157 is this: an unusually robust preclinical literature has established a plausible and broadly consistent mechanistic picture, while the human clinical trial evidence remains essentially absent
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