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DNA double-strand breaks (DSBs) activate ATM and ATR kinases, initiating a cascade that leads to phosphorylation of signaling proteins such as Nijmegen breakage syndrome (NBS1) and the histone variant H2AX (H2AX), ultimately resulting in p53 activation and transactivation of CDK inhibitors (Lscarez-Lagunas et al., 2022)

There are no enzymes in the RBC that catalyze the reduction of NAD + by NADPH, so that high levels of both NAD + and NADPH can exist simultaneously in the same compartment

Hence, it was notably linked to clinicopathological features of HCC, including AFP levels, tumor grade, and tumor size [95]