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Borroz et al., 1994), 5,10-dihydroindeno[1,2- b ]indole and tert -butyl hydroquinone (TBH) (Liu 1994 and 1996, Yang et al., 2002), zinc (Ha et al., 2006), melatonin (Urata et al., 1999), curcumin (Zheng et al., 2007), formation of Michael reaction acceptors (containing an electrophilic electron-deficient center that is susceptible to nucleophilic attack) by treatment with diethyl maleate (DEM) or phorone to produce GSH conjugates (Borroz et al., 1994
Prior authorisation has become one of the clearest examples of this structural burden
However, ESPEN states that zinc supplementation may improve dysgeusia and indirectly support nutritional intake, and that restoring subnormal plasma levels can enhance urea production [11]