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BPC-157 handles the injury site

Gastrointestinal studies have shown that orally administered BPC-157 exerts cytoprotective effects throughout the digestive tract, including protection against NSAID-induced gastric lesions, alcohol-induced mucosal damage, and stress-induced intestinal barrier dysfunction

its blockade increases serotonin release Promotes neurogenesis : TREK-1 knockout mice show increased hippocampal neurogenesis, and PE-22-28 recapitulates this effect pharmacologically within 4 days of treatment Induces synaptogenesis : TREK-1 blockade promotes dendritic growth and synaptic connection formation PE-22-28 inhibits TREK-1 with an IC50 of 0.12 nM, making it approximately 300-500 times more potent than spadin