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Depleted glutathione by NAPQI leads to increased H 2 O 2 release that oxidizes thioredoxin and induces the disassociation of thioredoxin from apoptosis signalling-regulating kinase 1 (ASK-1), triggering self-activation of ASK-1, which phosphorylates mitogen-activated protein kinase kinase 4/7 and activates c-Jun N-terminal kinase (JNK) (Nakagawa et al., 2008)

Binding activates adenylate cyclase through Gs alpha coupling, elevating intracellular cyclic AMP and triggering the release of stored growth hormone (GH) from secretory granules

Softic et al., 2016)

Targeting metabolic cross talk between cancer cells and cancer-associated fibroblasts