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The formation of self-propagating aggregates by the yeast prion proteins generally leads to a loss-of-function of the native protein and subsequently the appearance of a novel phenotype, which can be transmitted to daughter cells and meiotic progeny by cytoplasmic inheritance (Liebman and Chernoff, 2012

Journal of Viral Hepatitis

Conversely, given the pro-oxidant and pro-inflammatory effects of excess copper within plaques, preclinical studies have reported that strategies utilizing copper chelators may hold potential for attenuating plaque progression, although this requires further investigation