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The role of copper in this regard is argued to be protective, with copper being released into the synaptic cleft at concentrations of up to 100 M upon depolarization, inhibiting the activity of postsynaptic NMDA, AMPA, and GABA receptors upon binding, thus preventing overstimulation in a negative feedback system (DAmbrosi and Rossi, 2015)

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260,261 Furthermore, treatment with a histone deacetylase inhibitor in Shank3 -knockout mice significantly improves the behavioural phenotype of the mice, suggesting that abnormal histone modification is a potential mechanism of ASD

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