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In mouse models of highfat dietinduced liver steatosis, it significantly inhibits hepatic NNMT activity, reduces NAM methylation, increases NAD+ and SAM levels, enhances mitochondrial fatty acid oxidation, reduces hepatic triglyceride and lipid accumulation, lowers lipotoxicity markers (e.g., malondialdehyde, transaminases), improves hepatocellular injury, and reverses steatosis

MTHFR (methylenetetrahydrofolate reductase) is an enzyme that helps your body convert folate and homocysteine into usable forms

Therefore, awareness of these signs and timely medical assessment are crucial for optimal outcomes