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Altogether, these data suggest that the upregulation of GSH, rather than the dTTP pool, is responsible for avoiding oncogene-induced replication stress and less susceptibility to JMF4073 in T315I-Bcr-Abl-32D cells

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J Nutr 143: 1726, 2013 [DOI] [PMC free article] [PubMed] [Google Scholar] 15.Evans JL, Maddux BA, and Goldfine ID

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