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When this genomic breadth is cited to justify systemic GHK-Cu dosing the reasoning being that a molecule touching thousands of genes must be doing something profound throughout the body it is worth remembering that broad gene modulation is as plausibly a reason for caution as for enthusiasm, since specificity, not breadth, is usually what separates a useful therapeutic from an indiscriminate one

This mechanism is distinct from growth hormone secretagogues, incretin mimetics, or direct receptor agonistsmeaning AHK-Cu doesn't compete for the same binding sites as GLP-1 analogs, growth hormone releasing peptides, or melanocortin receptor targets

Balanced hormones are essential for ovulation in individuals with ovaries and sperm production in individuals with testes

A lot of conventional doctors think this is all bullshit, he told me, smiling