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Cold shock evoked transient increases in [Ca2+cyt] level in tobacco and Arabidopsis (Knight et al., 1991

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The potential sources for the ROS include hepatic cytochrome P450 2E1, mitochondria and iron overload.7 Mitochondrial damage also produce ROS which may trigger steatohepatitis and fibrosis by lipid peroxidation, cytokine induction and induction of Fas ligand.5 Excessive fatty acid oxidation is a major contributor to oxidative stress in the liver

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