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The accumulation of harmful metabolites and depletion of protective metabolites contribute to fibrosis progression through various mediators, including the renin-angiotensin system, reactive oxygen species, Toll-like receptor 4, aryl hydrocarbon receptor, inhibitor of kappa B/nuclear factor kappa B, and Kelch-like ECH-associated protein 1/nuclear factor erythroid 2-related factor 2 pathways

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It is often paired with regenerative therapies, such as stem cell treatments, to enhance recovery and outcomes

Accordingly, it is crucial to understand that fecal SCFA excretion does not entirely reflect their concentration and production within the gut