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2 Ammonium metabolism in tumor cells: sources, fates, and functional implications 2.1 Metabolic reprogram and crosstalk of ammonium metabolism in prostate cancers PCa cells exhibit distinct ammonium metabolism characterized by dysregulated upstream amino acid catabolism and coordinated crosstalk with glucose and lipid metabolic pathways

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However, NAD precursor treatment alone failed to raise the decreased levels of GSH seen in 3xTg-AD neurons, as much as a combination of nicotinamide and a redox-regulating Nrf2-inducer was able to restore GSH above nTg levels [71]