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Additional studies have explored activation of focal adhesion kinase (FAK) and paxillin pathways in fibroblasts, which are involved in cellular migration, extracellular matrix interactions, and structural tissue dynamics [2]

Despite this strength, the tendon remains vulnerable to both acute rupture and chronic degenerative conditions

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Research on AHK-Cu and Hair Growth Studies suggest that AHK-Cu may stimulate the proliferation of dermal fibroblasts, which produce substances like VEGF, essential for blood vessel growth[2]