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Conclusion Our research provided strong evidence to support the conclusion that HOXA2 expression was inhibited by aberrant hypermethylation, leading to decreased SIRT1 levels, which could increase ATF6 acetylation and activation as well as subsequent ER stress, resulting in renal damage in CKD-related fibrosis

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However, it decreased the activity of glutathione-S-transferase in the liver and small intestine

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