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Connecting phenotype to genotype: searching for the genetic basis of efficient arsenic metabolization Given the above phenotype, different genes encoding reductases and methyltransferases have been hypothesized to be the source of the inter-individual and inter-population variation in arsenic susceptibility (Fujihara et al

Other study proved that nicotine applied subcutaneously in a daily dose of 1.5 mg/kg for 4 weeks may damage rat lung tissue and may lead to the intraparenchymal haemorrhage, respiratory epithelial proliferation, extensive interstitial fibrosis and empchysematous changes (3)