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These data suggest that, in addition to GR1-dependent depletion of GSH, dexamethasone modulates additional mechanisms of ferroptosis resistance that are independent of both GPX4 and FSP1

The FITC-dextran assay demonstrated that the addition of QCT significantly attenuated the increased endothelial cell permeability caused by Erastin (Fig

[,] Moreover, GSH plays a major role in protecting important cell constituents from the destruction caused by ROS such as free radicals, peroxides, and lipid peroxides

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