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[1] At the cellular level, the tendon work is genuinely mechanistic: in cultured rat tendon fibroblasts, BPC-157 accelerated cell outgrowth, improved survival under oxidative stress, and increased migration in a dose-dependent way, apparently through the FAKpaxillin pathway

The first improvements often appear in hair texture and strength rather than immediate length increases

NAC supports glutathione production, helping to reduce oxidative stress, calm inflammation, and assist with estrogen detoxification

Both systemic injection (intramuscular) and local application (directly at the bone defect) produced healing improvements