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Effect of ginger and L-carnitine on the reproductive performance of male rats

21 This largely underlies why defective mitophagy has been implicated in synaptic dysfunction in the early pathophysiology of PD

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PURINE CATABOLISM In addition to GSH redox coupling mechanism and NO signaling, purine catabolism ( Figure 2 ) may be a previously unappreciated component of the homeostatic response of mitochondria to oxidant stress and may play a critical role in slowing progressive mitochondrial dysfunction in certain disease states (Kristal et al., 1999)