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Accordingly, these principles have been therapeutically exploited in nanomedicine platforms designed to induce GSH-dependent ROS amplification (Fig

262 In addition, transgenic expression of PGC-14 in skeletal muscle induces VEGF in vivo, whereas the knockdown of PGC-14 abrogates the induction of angiogenesis in response to hypoxia

LEAP2 as a therapeutic target in obesity and cardiometabolic disorders

The presence of multiple hgp genes may allow for selective expression of the Hgp with greatest affinity for the predominant host haemoglobin-haptoglobin phenotype, or alternatively as a strategy to evade the host immune response mounted against a particular Hgp-expressing population (Langlois and Delanghe, 1996)